Kathleen Potter, DVM, PhD
Primary current research interest is in discovering and reporting
new diseases in animals via diagnostic pathology. Principal funded
research projects in the past have involved pathogenesis of eosinophilic
inflammation associated with Taenia taeniaeformis infection; and
pathogenesis and comparative pathology of bovine Marfan syndrome.
Collaborative research projects have included analysis of trefoil
peptide expression in disease of dogs and cats (Bonnie Campbell,
Principal Investigator) pathogenesis of pneumonia Rocky Mountain Bighorn
Sheep (Subramaniam Srikumaran, Principal Investigator); and
pathogenesis of E. coli O157:H7 virulence factors (Tom Besser, Principal
Besser, TE, Cassirer EF, Potter, KA, et al.
Association of Mycoplasma ovipneumonia infection with
population-limiting respiratory disease in free ranging Rocky
Mountain bighorn sheep (Ovis canadensis canadensis). J Clin
Lim, JY, Li J, Sheng H, Besser T, Potter, K, and
Hovde C. Colonization of E. coli O157:H7 at the recto-anal
junction of long duration culture positive cattle. Applied
and Environmental Microbiology 73:1380-1382, 2006.
Singleton, AC, Mitchell, AL, Byers ,PH, Potter, KA
and Pace, JM. Bovine model of marfan syndrome results from an
amino acid change (E1200K) in a calcium-binding epidermal growth
factor-like domain of fibrillin-1. Human Mutation 25:348-352, 2005.
Gigante, A, Chillemi, C, Potter, KA,
Bertoni-Freddari, C, and Greco, F. Elastic fibers of musculoskeletal
tissues in bovine Marfan syndrome: a morphometric study. J. Orthpaed.
Res. 17:624, 1999.
Masabanda, J, Ewald, D, Buitcamp, J, Potter, K and
Fries, R. Molecular markers for the vovine fibrillin 1 gene (FBN1)
map to 10q26. Animal Genetics 29:450, 1998.
Pessier, AP, and Potter, KA. Ocular pathology in
bovine Marfan syndrome with demonstration of altered fibrillin
immunoreactivity in explanted ciliary body cells. Lab. Invest