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Our
work is focused on localization of these metabolic receptor cells and on tracing the
neural pathways from these receptors to parts of the brain which organize feeding
behavior. Understanding the basic anatomy and physiology of these particular metabolic
controls of food intake will contribute importantly to the overall view of how the nervous
system integrates feeding behavior, metabolism and body weight and ultimately achieves
metabolic homeostasis.
Biographical Information
Sue Ritter, Professor,
received an undergraduate degree in psychology from Valparaiso University
(1968) and a Ph.D. in physiological psychology from Bryn Mawr College (1973).
She joined W.S.U.s faculty in 1974.
I Wondered as I Wandered,
by Sue Ritter, third in The Human Side of Science series.
Selected Publications
Ritter, S.,
N.Y. Calingasan, B. Hutton and T.T. Dinh. 1992. Cooperation of central and peripheral
neural systems in monitoring metabolic events controlling feeding behavior. In: Neuroanatomy
and Physiology of Abdominal Vagal Afferents, S. Ritter, R.C. Ritter and C.D. Barnes
(Eds). CRC Press, Boca Raton, FL. 249-277.
Ritter, S., and T.T. Dinh. 1994. 2-Mercaptoacetate and
2-deoxy-d-glucose induce Fos-like immunoreactivity in rat brain. Brain Res. 641:
111-120.
Singer, L.K. and S. Ritter. 1994. Differential effects of
infused nutrients on 2DG- and MA-induced feeding. Physiol. Behav. 56: 193-196.
Singer, L.K. and S. Ritter. 1996. Intraventricular glucose
blocks feeding induced by 2-deoxy-D-glucose but not mercaptoacetate. Physiol. Behav.
59: 921-923.
Ritter, S., L.K. Singer, and A. Scheurink. 1995.
2-Deoxy-d-Glucose but not mercaptoacetate increases Fos-like immunoreactivity in adrenal
medulla and sympathetic preganglionic neurons. Obesity Res. 3(5): 729S-734S.
Ritter, S., and B. Hutton. 1995. Mercaptoacetate-induced
feeding is impaired by central nucleus of amygdala lesions. Physiol. Behav. 58:
1215-1220.
Koegler, F.H. and S. Ritter. 1996. Feeding induced by
pharmacological blockade of fatty acid metabolism is selectively attenuated by hindbrain
injections of the galanin receptor antagonist, M40. Obesity Res. 4: 329-336.
Singer, L.K., P. Magluyan, and S. Ritter. 1996. The effects
of low, medium and high fat diets on 2-deoxy-d-glucose (2DG)- and mercaptoacetate
(MA)-induced feeding. Physiol. Behav. 60: 321-323.
Ritter, S., Llewellyn-Smith, I., Dinh, T.T. 1998. Subgroups
of hindbrain catecholamine neurons are selectively activated by 2-deoxy-D-glucose induced
metabolic challenge. Brain Research 805: 41-54
Fraley, G.S., and S. Ritter. 2003. Immunolesion of norephinephrine and
epinephrine afferents to medial hypothalamus alters basal and 2DG-induced
NPY and AGRP mRNA expression in the arcuate nucleus. Endocrinology
144:75-83.
Ritter, S., A.G. Watts, T.T. Dinh, G. Sanchez-Watts, and C. Pedrow. 2003.
Immunotoxin lesion of hypothalamically-projecting norepinephrine and
epinephrine neurons differentially effects circadian and
stressor-stimulated corticosterone secretion. Endocrinology 144
(4): 1357-1367.
I'Anson H., Sundling L.A., Roland S.M., Ritter S. 2003. Immunotoxic
destruction of distinct catecholaminergic neuron populations disrupts the
reproductive response to glucoprivation in female rats. Endocrinology
144: 4325-4331.
PubMed Publications (Note: PubMed
Search may produce additional "S Ritter" authors.)
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